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Sawsan Al-Sinani, Departments of Biochemistry,College of Medicine & Health Sciences, Sultan Qaboos University, Muscat, Oman
Mohammed Al-Shafaee, Departments of Family Medicine & Public Health, College of Medicine & Health Sciences, Sultan Qaboos University, Muscat, Oman
Ali Al-Mamari, Departments of Medicine, Sultan Qaboos University Hospital, Muscat, Oman
Nicolas Woodhouse, Departments of Medicine, College of Medicine & Health Sciences, Sultan Qaboos University, Muscut Oman
Omayma El-Shafie, Departments of Medicine, Sultan Qaboos University Hospital, Muscat, Oman
Mohammed O. Hassan, Departments of Physiology, College of Medicine & Health Sciences, Sultan Qaboos University, Muscut, Oman
Said Al-Yahyaee, Departments of Genetics, College of Medicine & Health Sciences, Sultan Qaboos University, Muscut, Oman
Sulayma Albarwani, Departments of Physiology, College of Medicine & Health Sciences, Sultan Qaboos University, Muscut, Oman
Deepali Jaju, Departments of Clinical Physiology, Sultan Qaboos University Hospital, Muscat, Oman
Khamis Al-Hashmi, Departments of Medicine, College of Medicine & Health Sciences, Sultan Qaboos University, Muscut, Oman
Mohammed Al-Abri, Departments of Clinical Physiology, Sultan Qaboos University Hospital, Muscat, Oman
Syed Rizvi, Departments of Family Medicine & Public Health, College of Medicine & Health Sciences, Sultan Qaboos University, Muscat, Oman
Riad Bayoumi, Departments of Biochemistry,College of Medicine & Health Sciences, Sultan Qaboos University, Muscat, Oman

Abstract

Objectives: The aim of this study was to estimate the prevalence of impaired fasting glucose (IFG) among Omani adults with no family history (FH) of diabetes and to investigate the factors behind the risk of developing type 2 diabetes (T2D), while excluding a FH of diabetes. Methods: A total of 1,182 Omani adults, aged ≥40 years, visited the Family Medicine & Community Health Clinic at Sultan Qaboos University Hospital, Oman, on days other than the Diabetes Clinic days, from July 2010 to July 2011. The subjects were interviewed and asked if they had T2D or a FH of T2D. Results: Only 191 (16%) reported no personal history of T2D or FH of the disease. Of these, anthropometric and biochemical data was complete in 159 subjects. Of these a total of 42 (26%) had IFG according to the American Diabetes Association criteria. Body mass index, fasting insulin, haemoglobin A1C and blood pressure (BP), were significantly higher among individuals with IFG (P <0.01, P <0.05, P <0.01 and P <0.01, respectively). In addition, fasting insulin, BP and serum lipid profile were correlated with obesity indices (P <0.05). Obesity indices were strongly associated with the risk of IFG among Omanis, with waist circumference being the strongest predictor. Conclusion: Despite claiming no FH of diabetes, a large number of Omani adults in this study had a high risk of developing diabetes. This is possibly due to environmental factors and endogamy. The high prevalence of obesity combined with genetically susceptible individuals is a warning that diabetes could be a future epidemic in Oman.

Publication Date

7-4-2014

First Page

183

Last Page

189

Creative Commons License

Creative Commons Attribution 4.0 International License
This work is licensed under a Creative Commons Attribution 4.0 International License.

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